Answer :
The concentration of 2,3-BPG in erythrocytes "substantially increases" as people ascend to high altitudes, and this elevation is associated with a greater ability of hemoglobin to release oxygen.
The molecular mechanism underlying maximum elevation and disease-related hypoxia-induced erythrocyte 2,3-BPG remains unknown. However, it is still unclear what molecules underlie the hypoxia-related high altitude and disease-related erythrocyte 2,3-BPG.
We enlisted 21 young, healthy human volunteers and carried out high-altitude investigations to answer these questions. Our human research revealed that high altitude dramatically boosted erythrocyte 2,3-BPG and O2 releasing capacity while also inducing circulating adenosine levels and the activity of soluble ecto-5'-nucleotidase (CD73), a critical enzyme to produce extracellular adenosine.
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